Ben’s Health Log

Last updated: March 12, 2026

Profile

Diagnosis (Dec 2025)

Metabolic syndrome / prediabetes:

Bloodwork History

Marker Jul ’25 Oct ’25 Dec ’25 Mar ’26 Trend
Insulin (uIU/mL) 25.4 20 29.6 25.4 ↔ net, ↓ from Dec
Triglycerides 361 143 ↓↓↓ 🎉
A1c (%) 6.0 5.6 6.0 5.8
Glucose (mg/dL) 95 95 101 110 ↑ (see note)
ALT (U/L) 70 46 47 39 ↓↓ best ever
Creatinine 1.08 1.3 1.4 1.29 ↓ (was creatine artifact)
eGFR (creatinine) 94 74 68 75
eGFR (cystatin C) 84 new baseline
Ferritin 535 95 152 normalized
Uric Acid 7.9 7.0
HDL 38 39 ↔ (needs exercise)
C-Peptide 2.93 new baseline

Glucose note: Fasting glucose paradoxically higher (101→110) while A1c improved. Classic OMAD pattern — hepatic glucose output increases during long fasting windows (dawn phenomenon), but average glucose is lower as confirmed by A1c and CGM data.

Kidney — CLOSED: eGFR decline (94→68) was entirely due to ~10g/day creatine supplementation confounding creatinine-based eGFR. Cystatin C eGFR = 84 (normal). Creatine discontinued. No kidney concern.

Current Protocol (Lent 2026: Feb 18 – Apr 5)

Daily: OMAD

Extended Fasts (3-day water fasts)

Exercise

Tracking

What Worked

  1. 3-day fasts are the primary lever. Per-fast modeling: trigs drop ~37% multiplicatively, insulin drops ~7.4%. Trigs went from 361→143 with 2 fasts + 3 weeks OMAD.
  2. OMAD is effective maintenance between fasts. Sustains the metabolic pressure.
  3. Exercise stacking (Z2 AM + KB PM) produces clean glycogen depletion curves on CGM. March 9 was best day: glucose 110→89 through stacked exercise, dinner peaked at 146.
  4. Fasted Z2 cycling depletes glycogen efficiently. TTGNG around 8am confirms liver glycogen clearing by mid-morning.
  5. Weight loss: 239→227 (~12 lbs) in first 2 weeks.

What Didn’t Work

  1. Dietary keto (eating keto, not fasting): 4+ days of keto eating produced only 0.1 mmol/L BHB. Insulin too high for dietary keto to produce meaningful ketones. Abandoned.
  2. Big carb OMAD: Feb 25 baptism anniversary dinner (noodles, dumplings, corn) caused massive glucose spike, poor sleep, felt hungover. High-glycemic OMAD is counterproductive.
  3. Creatine supplementation: 10g/day confounded eGFR readings for months, creating false kidney alarm. Discontinued.

Failed Experiments

Modeling & Projections

Per-fast decay model (from 2 data points, heavy caveats):

Diminishing returns expected:

Early fasts clear easy liver fat. As metabolic health improves, per-fast yield shrinks. Sub-5 insulin is a 2027 goal if it happens.

Phased Strategy

Phase 1 — NOW → ~Oct 2026: Break insulin resistance

Phase 2 — ~Oct 2026 → Spring 2027: Rebuild metabolic flexibility

Strength training can start now

North Star Metric

Morning fasting BHB (blood ketones). Inverse proxy for insulin. Target: >0.5 mmol/L consistently on non-fast days. Currently 0.2 on normal OMAD days.

Research Knowledge Base

Data Files

Open Questions

  1. Will per-fast insulin drops accelerate or decelerate as liver fat clears?
  2. Is fasting glucose rise (101→110) concerning, or just dawn phenomenon from OMAD?
  3. When to add GGT to bloodwork panel? (proxy for liver fat)
  4. Should strength training start now or wait for Phase 2?
  5. What does C-Peptide 2.93 tell us about beta cell function?